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Abstracts
L.E. Norton, M.R. Distasi, R.G. Bills
and J.L. Unthank. Impairment of collateral artery growth in the
Spontaneously Hypertensive Rat (SHR) is mediated via reactive oxygen
species (ROS). FASEB J. 19(4):A1271, 2005.
L.E. Norton, M. R. Distasi, R.G.
Bills, and J.L. Unthank. Therapeutic enhancement of collateral
artery growth in the Spontaneously Hypertensive Rat (SHR). Vascular
Medicine 2005; 10:137:P-7.
L.E.
Norton, M.R. Distasi, R.G. Bills, J.L. Unthank, M.C. Dalsing.
Collateral artery growth in the spontaneously hypertensive rat (SHR) is
impaired by NADPH oxidase. Journal of the American College of Surgeons
201:S99, 2005.
M.R.
Distasi, L.E. Norton, R.G. Bills, T.L. Haas, and J.L. Unthank. Matrix
metalloproteinase inhibition limits luminal expansion but not
endothelial proliferation in rat collateral arteries. FASEB J.
19(4):A1271, 2005.
S.J.
Miller, K.A. Kerr, W.C. Watson, C.L. Morrison, L.E. Norton, J.L.
Unthank. Molecular basis for altered regulation of reactive oxygen
species and nitric oxide in impaired collateral growth in the SHR.
Microcirculation 12:684-5, 2005.
L.E.
Norton, M.R. Distasi, M.C. Dalsing, S.J. Miller, J.L. Unthank.
Impairment of collateral growth in spontaneously hypertensive rats: A
role for NADPH oxidase? FASEB J. 20(5):A1099, 2006.
S. J.
Miller, X. Zhou, L. E.
Norton, M. P. Murphy, M. C. Dalsing, H. G. Bohlen, and J. L. Unthank.
Regulation of local angiotensin type 1b receptor (AT1bR)
expression explains collateral growth impairment in spontaneously
hypertensive rats (SHR) and restoration of collateral growth (CG) by
captopril. FASEB J: 22: 1147.7., 2008
Awards
Junior
Scientist Award (Postdoctoral and Resident Category), From
North American Vascular Biology Organization for Presentation at
Experimental Biology Meeting in San Francisco, April, 2006
Presentations
Norton
LE, Distasi MR, Dalsing MC, Miller SJ, Unthank JL. Impairment of
collateral growth in spontaneously hypertensive rats: A role for NADPH
oxidase? North American
Vascular Biology Organization: Highlights in Trainee Research and
Welcome Reception.
Experimental Biology 2006. San Francisco, CA April 2006
S.
J. Miller, X. Zhou, L. E.
Norton, M. P. Murphy, M. C. Dalsing, H. G. Bohlen, and J. L. Unthank.
Regulation of local
angiotensin type 1b receptor (AT1bR) expression explains collateral
growth impairment in spontaneously hypertensive rats (SHR) and
restoration of collateral growth (CG) by captopril.
Cardiovascular
Section Featured Topic: Arteriogenesis and Vascular Remodeling.
Sponsored by the American Physiological Society at Experimental Biology
2008. San Diego, CA April 8, 2008.
Publications
Miller S.J.,
L. E. Norton, J. Theordorakis, M. P. Murphy, M. C. Dalsing, and J. L.
Unthank. Role of the Renin-Angiotensin System and NAD(P)H Oxidase in the
Impairment of Collateral Growth in the SHR. Am. J. Physiol.
292(5):H2523-31, 2007.
T. L. Haas, J. L. Doyle, M. R. Distasi, L. E. Norton,
K.
Sheridan, and J.L. Unthank. Role of MMPs in
outward remodeling of collateral mesenteric arteries. Am. J.
Physiol. 293: H2429 - H2437, 2007
Norton LE, Komenaka IK, Emerson RE, Murphy C, Badve S.
Benign glandular inclusions a rare cause of a false positive sentinel
node. J
Surg Oncol. 95:593-6,
2007.
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